Learning Objectives: 

  1. Learn how a non-synonymous coding sequence polymorphism in the KCNK16 gene may result in an increased risk of type-2 diabetes by increasing the activity of TALK-1 potassium channels.
  2. Learn how two-pore-domain potassium channels provide a countercurrent for endoplasmic reticulum (ER) calcium release to modulate hormone secretion and the ER stress response.


Session date: 
11/06/2017 - 5:00pm to 6:00pm CST
UCMC M-170
Chicago, IL 60637
United States
  • 1.00 AMA PRA Category 1 Credit™
  • 1.00 Participation

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Speaker Name: 
David A. Jacobson, Ph.D.